An international team of researchers has identified, for the first time, the cell types, areas and biological processes in the brain that mediate the genetic risk of insomnia. This was made possible by assessing DNA and sleep features in no less than 1.3 million people. The findings are a major step towards getting grip on the biological mechanisms that cause insomnia.
Children sleep more poorly if their mothers suffer from insomnia symptoms – potentially affecting their mental wellbeing and development.
According to researchers, increasing levels of Bmal1 in the skeletal muscles of mice makes the animals resistant to insomnia and sleep deprivation. The findings could provide a new treatment target for sleep disorders.
Older adults whose lives have meaning enjoy better sleep quality and less sleep apnea and restless leg syndrome.